primary antibody rat anti–mouse mac2 Search Results


lgals3 antibodies  (Miltenyi Biotec)
92
Miltenyi Biotec lgals3 antibodies
Lgals3 Antibodies, supplied by Miltenyi Biotec, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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rat anti mac2  (Cedarlane)
93
Cedarlane rat anti mac2
Rat Anti Mac2, supplied by Cedarlane, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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purified anti-mouse mac-2  (Cedarlane)
90
Cedarlane purified anti-mouse mac-2
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Purified Anti Mouse Mac 2, supplied by Cedarlane, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
purified anti-mouse mac-2 - by Bioz Stars, 2026-03
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m3 38  (Cedarlane)
96
Cedarlane m3 38
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
M3 38, supplied by Cedarlane, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 96 stars, based on 1 article reviews
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monoclonal rat anti-mouse antibody anti-mac-2  (Burlington Industries)
90
Burlington Industries monoclonal rat anti-mouse antibody anti-mac-2
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Monoclonal Rat Anti Mouse Antibody Anti Mac 2, supplied by Burlington Industries, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 90 stars, based on 1 article reviews
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mouse anti galectin 3 mab  (Proteintech)
94
Proteintech mouse anti galectin 3 mab
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Mouse Anti Galectin 3 Mab, supplied by Proteintech, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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mouse anti galectin 3 mab - by Bioz Stars, 2026-03
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anti mac 2  (Cedarlane)
94
Cedarlane anti mac 2
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Anti Mac 2, supplied by Cedarlane, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti mac 2/product/Cedarlane
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rat anti mac 2  (Cedarlane)
92
Cedarlane rat anti mac 2
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Rat Anti Mac 2, supplied by Cedarlane, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/rat anti mac 2/product/Cedarlane
Average 92 stars, based on 1 article reviews
rat anti mac 2 - by Bioz Stars, 2026-03
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mac2  (Cedarlane)
93
Cedarlane mac2
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Mac2, supplied by Cedarlane, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Average 93 stars, based on 1 article reviews
mac2 - by Bioz Stars, 2026-03
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mac 2  (Cedarlane)
94
Cedarlane mac 2
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Mac 2, supplied by Cedarlane, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti mouse mac 2 antibody  (Cedarlane)
91
Cedarlane anti mouse mac 2 antibody
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Anti Mouse Mac 2 Antibody, supplied by Cedarlane, used in various techniques. Bioz Stars score: 91/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti mouse mac 2 antibody - by Bioz Stars, 2026-03
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mouse monoclonal anti galectin 3  (Santa Cruz Biotechnology)
95
Santa Cruz Biotechnology mouse monoclonal anti galectin 3
RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and <t>macrophage</t> (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.
Mouse Monoclonal Anti Galectin 3, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and macrophage (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.

Journal: bioRxiv

Article Title: Resolvin D2/GPR18 signaling enhances monocytic myeloid-derived suppressor cell function to mitigate abdominal aortic aneurysm formation

doi: 10.1101/2024.02.23.581672

Figure Lengend Snippet: RvD2 administration decreases aortic aneurysm phenotype and preserves morphology to mitigate AAA formation. A , Schematic description of murine topical elastase model with RvD2 treatments. Mice were divided into three groups and treated with either heat-inactivated elastase or elastase on day 0. Mice were then administered either vehicle or RvD2. Aortic diameter was measured on day 14 and tissue harvested for additional analysis. B , RvD2 treated mice demonstrated a significant decrease in aortic diameter compared to vehicle treated mice; *p<0.001 vs. other groups; n=10-20 per group). C , Representative images of aortic phenotype in the respective groups. D , Comparative histology performed on day 14 indicates that elastase-treated mice administered with RvD2 have a marked increase in smooth muscle cell α-actin (SM-α actin) expression, decrease in elastic fiber disruption (Verhoeff-Van Gieson staining for elastin) as well as neutrophil (PMN) and macrophage (Mac-2) infiltration, compared to elastase-treated WT mice alone (n=5 per group). Representative histological images in the respective groups with arrows indicate areas of immunostaining. E , Quantification of histological staining in respective groups; n=5 per group; *p<0.02 vs. other groups.

Article Snippet: Slides of aortic cross-sections were prepared and stained for elastin (Van Gieson’s Solution, catalog no. s289; Poly Scientific R&D Systems, Bay Shore, NY) smooth muscle actin (monoclonal anti-actin α-smooth muscle, Sigma Aldrich, St. Louis, MO), macrophages (purified anti-mouse Mac-2, Cedarlane Laboratories, Burlington, Canada), and neutrophils (rat anti-mouse neutrophils, AbD Serotec, Oxford, UK).

Techniques: Expressing, Disruption, Staining, Immunostaining

In vivo GPR18 knockdown reduces the protective effect of RvD2. A , GPR18-siRNA treatment of WT mice demonstrated a significant increase in aortic diameter as compared to control (c)-siRNA treated mice after administration of RvD2 in respective groups (*p=0.004, n=12-13 per group). B , Representative images of aortic phenotype in respective groups. C, Expression of smooth muscle-α actin is significantly increased, and elastin fragmentation as well as macrophage and neutrophil infiltration in aortic tissue are significantly decreased in mice treated with c-siRNA+RvD2 compared to mice treated with GPR18 siRNA+RvD2 (n=5 per group). Arrows indicate areas of immunostaining. D, Quantification of histological staining in respective groups; n=5 per group; *p<0.01 vs. other groups.

Journal: bioRxiv

Article Title: Resolvin D2/GPR18 signaling enhances monocytic myeloid-derived suppressor cell function to mitigate abdominal aortic aneurysm formation

doi: 10.1101/2024.02.23.581672

Figure Lengend Snippet: In vivo GPR18 knockdown reduces the protective effect of RvD2. A , GPR18-siRNA treatment of WT mice demonstrated a significant increase in aortic diameter as compared to control (c)-siRNA treated mice after administration of RvD2 in respective groups (*p=0.004, n=12-13 per group). B , Representative images of aortic phenotype in respective groups. C, Expression of smooth muscle-α actin is significantly increased, and elastin fragmentation as well as macrophage and neutrophil infiltration in aortic tissue are significantly decreased in mice treated with c-siRNA+RvD2 compared to mice treated with GPR18 siRNA+RvD2 (n=5 per group). Arrows indicate areas of immunostaining. D, Quantification of histological staining in respective groups; n=5 per group; *p<0.01 vs. other groups.

Article Snippet: Slides of aortic cross-sections were prepared and stained for elastin (Van Gieson’s Solution, catalog no. s289; Poly Scientific R&D Systems, Bay Shore, NY) smooth muscle actin (monoclonal anti-actin α-smooth muscle, Sigma Aldrich, St. Louis, MO), macrophages (purified anti-mouse Mac-2, Cedarlane Laboratories, Burlington, Canada), and neutrophils (rat anti-mouse neutrophils, AbD Serotec, Oxford, UK).

Techniques: In Vivo, Knockdown, Control, Expressing, Immunostaining, Staining